
Wound healing in the injured cord. Macrophages (CD11B+ cells) are abundant within vascular-rich (PECAM+) zones at both 7 and 14 days after contusion injury to the murine spinal cord.

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Principal Investigator
Professor of Neurological Surgery and of Physical Therapy and Rehabilitation
Alvera L. Kan Endowed Chair of Neurological Surgery
Co-Director and Principal Investigator, Brain and Spinal Injury Center (BASIC)
Co-Director, Neurobehavioral Core for Rehabilitation Research
Hita Adwanikar
Postdoctoral Fellow
PhD, Baylor College of Medicine
Determinants of recovery in the injured, immature brain
Adanma Ekeledo
Research Associate UCSF
BS, Harvard University
Strategies to improve recovery after spinal cord injury
Thomas Fandel
Research Specialist
MD, University of Aachen, School of Medicine, Germany
Stem cell strategies to improve neurogenic bladder dysfunction after spinal cord injury
Sang Mi Lee
Postdoctoral Fellow
PhD, Yonsei University, Seoul, Korea
L-selectin as a mediator of secondary damage in the injured spinal cord
Alpa Trivedi
Assistant Researcher/ Molecular Biologist
PhD, Wright State University
Angiogenesis and spinal cord injury
Haoqian Zhang
Postdoctoral Fellow
PhD, Nagoya University, Japan
Matrix metalloproteinases and wound healing in the injured spinal cord

Wound healing in the injured cord. Macrophages (CD11B+ cells) are abundant within vascular-rich (PECAM+) zones at both 7 and 14 days after contusion injury to the murine spinal cord.